A sequential fluid-solid interaction model of expanded coronary-shear thinning blood is developedaccounting for effect of both stent and vessel deformation on haemodynamics. The proposed computational model is employed to investigate the influence of stent link with different geometrical configurations ( S-，N-shape) andvascular stenosis rates ( 24%，40%，50%) on haemodynamics. Both induced equivalent stress during stentimplantation and wall shear stress by pulsating blood flow are obtained for biomechanical injury assessment withinatherosclerosis plaque. The numerical results indicate that the developed high equivalent stress is located at theinterface between the narrowest vascular lumen and stent link while high wall shear stress on the plaque withmaximal radical tissue prolapse. With increasing vascular stenosis rates，the experienced equivalent stress and timeaveraged wall shear stress remarkably increase leading to higher risk of plaque rupture. Compared with N-type stent，higher peak equivalent stress and wall shear stress are observed on the plaque for S-type stent implying a